Kaempferia Parviflora for better erections and, yes, a larger penis

By tongkatali.org
tongkataliorg3@gmail.com
Updated June 1, 2022

In its January 15, 2011 edition, the respected magazine Psychology Today surprised with a remarkable article, headlined "The Rare Truth About Penis Size - Are there safe, effective ways to increase penis size? Yes." [Source]

The admission is noteworthy because the mainstream media, of which Psychology Today is part, has long ridiculed concerns about penis size and, with a stiff upper lip, proclaimed: Size doesn't matter.

The article in Psychology Today, written by Michael Castleman of San Francisco, makes some concessions to the politically correct attitudes about penis size, but also states: "Of course, an extra inch couldn't hurt."

Is a large penis important? For people in the porn industry, it is. The porn industry has a reason for requiring large penises: consumer choice. If many porn viewers preferred cute little penises, the porn industry would hire accordingly.

The porn actor best known for a large penis was John Holmes. When first married, he reported a length of 10 inches. During his career, the claim was jacked up to 13, 14, even 16 inches. His penis size was never independently verified. Camera angles can do a great job to make objects appear exceptionally large. Holmes played in more than 500 films, and he died of AIDS.

In 2017, it was reported that Roberto Esquivel Cabrera, then 54, of Saltillo, Mexico, demanded an entry in Guinness World Records for having the longest penis, some 50 cm. It was also reported that since he was a teenager, he had been hanging weights on his penis to make it bigger. What descends in between Cabrera's legs is heavily bandaged. Even for an examination by a physician, he would not take off the dressing. But he agreed to a CT (computed tomography) scan (probably not knowing what to expect), and this imaging technique revealed that his penis was actually only some 6 inches long. What reached his knees was just a heavily elongated prepuce (foreskin).

Below some video footage of Cabrera and his half meter, graphic but not pornographic.

Fueled by comical fools like Roberto Cabrera, the mainstream published opinion normally ridicules the concern of men about penis size.

An example is the following remark of sex adviser Ruth Westheimer, quoted from the TIME cover story of May 4, 1998: “Even if a man has an erection from floor to ceiling and can keep it that way for an hour, it will not be pleasurable for a woman if he is not sexually literate.” (The first version of this Serge Kreutz article was published in 2001, and this quote is carried over for more than 20 years.)

But the issue is not to either have a large penis, OR to be sexually literate. The issue is to have a large functional penis AND to be sexually literate.

Published opinion may be different from the opinions held privately, even by people who publish. The publishing trade has its own rules on what information may be disseminated. The motto of the New York Times is: "All the news that’s fit to print." When Adolph S. Ochs came up with the slogan in 1897, he probably had this in mind: All the news that's not obscene. Nowadays it means: All the news that is politically correct.

An article mentioning penis size as an important attribute when assessing the qualities of a man as a lover, would not have passed Ochs' censorship because of obscenity. The angle is still not politically correct, and thus seldom published.

But for people privately, size matters.

It's not that penis size would be a principal factor in partner choice for most women. Most women anywhere in the world worry whether they are treated kindly by an intimate partner of superior physical strength. In cultures that look down on women who are not in a continuous relationship with a spouse, their first, women want fidelity. And then, there are economic calculations.

The most prominent scientific author validating the above commonsense assumptions is the University of Austin professor David Buss, an authority in evolutionary biology. Among his bestselling books are The Evolution of Desire: Strategies of Human Mating (1995) and The Dangerous Passion: Why Jealousy Is as Necessary as Love and Sex (2000).

But there are two aspects that should not be confused. One is female partner choice, and the other is what women find sexually attractive. For practical reasons, on a global scale, many women may opt for a partner they do not find sexually very attractive.

In Western societies, men are concerned about penis size, because they have learned from intimate experience that a large (not strangely huge) penis is sexually attractive to women.

It's an issue not unlike physical height. Women have a clear preference for tall men. In Western societies, let's say between 6 feet (183 cm) and 6 feet 4 inches (193 cm), less in societies with lower average height. In Western societies, women are less attracted to men that are only 5 foot (152 cm). On the other hand, women would usually find 7 feet (213 cm) height in a man not appealing. Statistically, a bell curve. The tallest person in history was the American Robert Wadlow at 8 feet, 11 inches (272 cm).

Men can sense what height women find attractive, in spite of published opinion avoiding the issue. Even the most powerful men in human history are not immune to a desire to be of an attractive stature. Josef Stalin, for example, was only 5 feet 4 inches (1.63 m) but Soviet photographers and photo retouchers made their efforts to make him look taller.

Stalin disliked the French wartime leader Charles de Gaulle, and that de Gaulle was an offensive 6 feet 5 inches (1.96 m) probably did not earn him any of Stalin's sympathy. The two only met once, on December 2, 1944, and there are only French photos of the event. De Gaulle was not invited to Yalta where, from February 4 to 11, 1945, Allied leaders decided on the post-WWII world.

Men learn from their female sexual partners that a large penis is a good thing.

While women experience orgasms the easiest through clitoral stimulation, for those with vaginal orgasms, proper penis parameters (sufficient size, sufficient rigidity, and sufficient staying power) are necessary. Sexual technique? Usually, women who prefer vaginal orgasms will apply their own technique.

But even when clitoral stimulation is preferred, the idea of large, functional penises occurs in the fantasies that are necessary to make clitoral stimulation an orgasmic success.

A 2013 Australian scientific study has assessed how three parameters determined how physically attractive women find certain men: height, shoulder-hip ratio (athletic build), and penis size. Source: Penis size interacts with body shape and height to influence male attractiveness

The scientist projected life-size images of men, generated by the computer program MakeHuman onto a screen. The images expressed three variables, height, shoulder-hip ratio, and flaccid penis size. All in all, there were 343 images, each of which was modified for a single variable. The images did not vary in facial features.

The images represented the body and genital shapes women would encounter in up to 95 percent of all real-life scenarios. 105 heterosexual women with an average age of 26 years participated in the study. 71.8 percent were of European ethnicity, 20.9 percent Asian, and 7.3 percent from elsewhere. Every woman was shown 53 images that represented the full range of variations. No interviewer was present during the rating sessions, and attractiveness scores were recorded anonymously.

The middle image below shows the average of all 343 modifications. The depiction on the left shows the shortest stature, the lowest shoulder to hip ratio, and the smallest penis. The largest values are represented on the right.

The results were clear. Women rated men with a tall stature more pleasing than men with a short stature, preferred men with an athletic build, and were more attracted to larger penises. Apart from the scoring, viewing times were measured for each image. Test participants spent little time looking at images that they dismissed as non-attractive (short obese men with small penises). Larger penis size was associated with longer viewing duration.

Penis physiology and biomechanics: Erections are facilitated by increased blood flow to the penis, and through trapping this blood in the corpora cavernosa and the corpus spongiosum. A man's body holds about 5 liters of blood. When donating blood, the Red Cross draws less than half a liter, and requires a donor to rest for half an hour.

An average erect penis with a length of 13 centimeters (5.1 inches) and a girth of 11.5 centimeters (4.5 inches) draws about 125 milliliters of blood.

The volume of a cylinder is determined as the square root of the radius times height times 3.14 (pi, a mathematical constant). The volume of a 13 cm penis with a girth of 11.5 cm (radius 1.83 cm) is 137 milliliters, slightly more than the estimated needed 125 milliliters of blood for an erection (1.83 x 1.83 x 13 x 3.14 = 137). If the length is increased to 26 cm (10.2 inches), and the girth to 23 cm (9 inches), the needed blood volume for an erection would be about 1 liter (3.66 x 3.66 x 26 x 3.14 = 1093).

People who lose 20 percent of their circulatory blood (about 1 liter for an average male) may suffer a hypovolemic shock and pass out. Go beyond that, and severe organ failure is a distinct possibility. Something has to give... and it's unlikely to be the systemic circulation. A penis of 10 inches maximum size may have more than half a liter of blood flowing through it, but the body cannot afford to shut off even close to 1 liter of blood, and thus there will be no rigidity. And indeed, as related by colleagues of his industry, Holmes' blood engulfed penis would not be a conventional erection but rather a kind of soft meatloaf.

That is for 10 inches. But claims circulated that Holmes had an erect penis of 16 inches length and 13 inches circumference. But, please, where is the blood supposed to come from for a 16 inches (40.6 cm) by 13 inches (33 cm) penis? More than 3 liters of blood would be needed (5.25 x 5.25 x 40.6 x 3.14 = 3513). A body attached to such an erection could not stay alive on 2 liters of remaining blood.

Because of biomechanical limitations to functional penis size, it's better not to have a 10-inch member. A length of 8 inches (20.3 cm) and a girth of 7 inches (17.8 m) may be maximum values at which functionality is maintained (2.83 x 2.83 x 20.3 x 3.14 = 510 milliliters of blood). Beyond that, there will be erectile dysfunction.

If 8 inches length and 7 inches girth are not enough, one may want to consider a horse dildo. They are Made in China.

Below some comments on penis size by a female reader, Ka, carried over from 2003:

Hello,

I stumbled on your website a few hours ago. I very much enjoyed your essays. It’s almost 5 am here (Toronto, Canada), and I just stopped reading.

I just keyed in “love” into my search engine…I don’t even know why (I guess I just wanted to focus on something sweet before bed). Your inclusion of our neurology was great. And your sincerity was refreshing. I’ll read a few more of your articles tomorrow night.

Now, I got a few questions:

1. You praise tongkat ali very much. I heard that it raises your testosterone levels through the roof (which I guess would explain irritability, and sexual desire). But do you know how it effects woman? (Not that I need help in the arousal department). Just curious.

2. In your essay: “Female Orgasm in Asia”, you spoke about the female orgasm…and so on. I need to say this (actually to all the men in the world): Please don’t act like you are on a mission in bed. There were plenty of men who were determined to give me an orgasm…and I just ended up faking it. Once it seems that the guy is on a mission…the magic is gone. For me, I can be immensely satisfied without an orgasm (if one happens: great!… but if it doesn’t…then leave it alone).

3. You also talked about penis size. Well: IT MATTERS! (At least to me). There was a time that if I liked a guy, and thought that there may be potential for a more serious relationship, I would take things slow, not sleep with him right away… bla bla… But now: I try to get him into bed quickly. I don’t want to start liking him… and so on… and then find out he is tiny down there. For me it doesn’t have to be huge, but it has to be at least average. And for me, it’s not a psychological thing. I want to feel filled! And if he is not at least average, then I don’t want to waste my time with him. I don’t want to marry a man that is small. He can give the best oral in the world… but if he can’t stuff me properly… then I’m gone. I don’t want to get emotionally attached, no matter how great they are, and then find out I’m gonna be cheating on them for sure if we continue.

Please don’t get me wrong: it doesn’t have to be mega huge… but at least average. I dated a few “small” men. Sometimes I wondered what the point of us having sex was. Cuz for me nothing beats the main course.

4. I don’t understand how increasing dopamine levels can help a man get erect. Doesn’t cocaine increase the amount of dopamine in your synapses? But… enough cocaine… and a man just can’t get it up. I don’t get it. (I dated a cocaine user…and there were soooooooooooooooooo many disappointments.)

Don’t feel like you have to rush with the answers.

Thanks,

Realistic penis size management

Many men would like to have a larger penis. Nothing wrong with that. Apart from a larger penis, many men would also like to have a more athletic physique, and, as we are at it, a more powerful brain. The question is: how far would one go TRYING to achieve these improvements. Anabolic steroids for the athletic physique as advocated in the Underground Body Opus by steroid guru Dan Duchaine, or LSD trips to expand one's mind as promoted in Turn On, Tune In, Drop Out by underground messiah Timothy Leary.

Or, much less metaphysical, going under the knife for some phalloplasty (cosmetic surgery of the penis).

But the abuse of anabolic steroids severely shortens a man's lifespan, and psychiatric hospitals around the world housed, for decades, thousands who became schizophrenic after a bad trip. The scientific term is: LSD Psychosis

The possible side effects of enlargement phalloplasty include a deformed penis and permanent erectile dysfunction, with a blood engorged penis just hanging down between the legs because the suspensory ligament was cut, which would normally provide an upward angle. Things can also go wrong in a more dramatic manner. On March 5, 2019, the Israeli billionaire diamond trader Ehud Arye Laniado died during a penis enlargement surgery in Paris. Two years earlier, a Swedish man died during penis enlargement surgery from injected fat getting dislodged into his blood circulation and causing an embolism.

Disfigurement (like Roberto Cabrera's prepuce reaching to his knees) and dysfunction will also be the only results of penis enlargement by hanging weights.

The only realistic approach is working on penile tissue tone.

Every man knows that on occasions, it is easy to have a larger penis. The penis doesn't have a fixed size. The size of the organ follows the tone of penile tissue. Depending on tissue tone, the size can increase substantially. In a standard setting, sexual arousal will initiate the appropriate biochemistry.

Positively toned penile tissue is going to be substantially bigger (double in size) than negatively toned penile tissue (maltreated with sympathetic drugs like cocaine or amphetamines, or even just too much coffee).

Penis size doesn't depend on the number of cells, but the constitution of cells. That's not unique to the penis but holds true for other organs, too.

Muscles, the kind bodybuilders train (they are called skeletal muscles), for example. Bodybuilders who are bulking up don't grow a single new muscle cell. They just enlarge existing muscle cells. Are the gains of bodybuilders permanent? No, of course not. Stop training, and over some not-so-long time, gains are lost.

The human penis is a sponge. For an erection to occur, the penis must fill with blood. The biochemical processes that decrease or increase penis size depend on blood vessels and the smooth muscle cells of erectile sponge tissue. There are several intertwined pathways by which blood flow to the penis, and the retaining of blood, is ruled. The sympathetic nervous system generates flaccidity impulses that contract penile tissue, while the parasympathetic nervous system relaxes the smooth muscle cells of the blood vessels and the sponges of the corpora cavernosa and corpus spongiosum, allowing engorgement. But this is not an either-or switch. Depending on many factors, engorgement and shrinkage are gradual. Flaccidity in one and the same penis can vary between 1 and 4 inches of length, and this is pharmacologically controllable, without sexual stimuli.

Sympathetic tissue tone is easily induced by amphetamines and amphetamine-mimicking drugs: speed, meth, ice, cocaine, crack, and other substances in this category will, dose-dependent, shrink the penis to amazingly minimal dimensions. Just abstaining from these substances of abuse will automatically double or triple penis size, when flaccid and when, sort-of, erect.

Minor sympathetic stimuli like coffee and caffeine-containing beverages such as energy drinks will have a similar, albeit lesser, effect.

Hormonal factors also play a role. Anything that decreases the functionality of the testes will also decrease the size of the testes and adjacent tissue. Exogenous testosterone analogues, such as anabolic steroids (most commonly Deca-Durabolin) or testosterone replacement therapy, will shut down the synthesis of testosterone by the testes and such cause partial atrophy (deca dick).

On the other hand, tongkat ali which enhances testicular function may cause an increase in testicular and penile size, though the effects are not as dramatic as the reverse through sympathetic stimulants. In bodybuilding circles, tongkat ali is an established testicular function restarter after steroid cycles.

When it comes to penis size and function, it is always easier to disturb processes than to enhance them.

For erectile function, there is only one FDA-approved category of medications, phosphodiesterase type 5 inhibitors (Viagra, Cialis, Levitra, and their generic equivalents). They all interfere with a course of events that starts with the conversion of L-arginine into nitric oxide by the enzyme nitric oxide synthase. Nitric oxide stimulates the enzyme soluble guanylate cyclase (sGC) to form cyclic guanosine monophosphate (cGMP) from guanosine triphosphate (GTP). The cGMP causes smooth muscle relaxation around the penis, resulting in the penis filling with blood, which then presents as an erection.

The effects of cGMP in the penis are kept in check by phosphodiesterases type 5, an enzymes that breaks down cGMP. Medications like sildenafil, tadalafil, and vardenafil work by interfering with, and inhibiting, the enzyme phosphodiesterase type 5, thus allowing cGMP to do its job for a prolonged time.

On a side note, many pharmaceutical drugs, including antibiotics, work by interfering with enzymes, either of the human body or of pathogens like bacteria. [See: A summary of drugs targeting enzymes]

The blockage of phosphodiesterase type 5 is not the only potential point of enzymatic interference to facilitate erectile function.

Another potential pathway would be the interference with the enzyme rho kinase, which, when overly expressed, contributes to the hardening of the walls of blood vessels and erectile tissue. Blood vessels and tissue with hardened cell walls cannot properly relax, even when flooded with cGMP as a result of phosphodiesterase type 5 inhibition. Incidentally, tongkat ali is a rho kinase inhibitor, and improves erectile function by helping to keep penile blood vessels elastic. No pharmaceutical rho kinase inhibitors for better erections are yet available.

Targeting arginase would yet another potential enzyme-blocking pathway to support erectile function. Arginase, which converts L-arginine into L-ornithine, is overly expressed in patients with hypertension, diabetes, obesity, and other modern diseases. Arginase competes with nitric oxide synthase for the same substrate, the amino acid L-arginine. The idea is that if arginase is inhibited, there will be more L-arginine available for nitric oxide synthase, which should result in more cGMP, and thus better erections. Alas, while there are pharmaceutical arginase inhibitors, none of them causes better erections or a more relaxed penile tissue tone.

And while dietary arginine supplementation is employed to improve the performance of livestock (semen quality, more meat upon slaughter), the data on arginine supplementation for human male sexual parameters is not encouraging.

On the other hand, many plants of the Zingiberaceae or ginger family (to which Kaempferia parviflora also belongs) have anti-arginase effecs, resulting in the amelioration of many ailments caused by aging, not just erectile dysfunction.

Nevertheless, the most usefulness, Kaempferia parviflora has as a low-affinity phosphodiestarase type 5 inhibitor. With prolonged use, the genital-shrinking effect of omnipresent phosphodiesterase will be reduced, thus contributing to a background relaxation of smooth muscle cells in the penis and allowing an improved flow of blood to the penile sponges. Obviously, when this happens, natural erections will be easier, and the penis will be enlarged, even when not being erect.

In sexual slang, Kaempferia parviflora will turn a "grower" (a small flaccid penis that grows substantially with an erection) into a "shower" (a large flaccid penis which, however, grows less dramatically when achieving an erection). "Shower", in this context, has nothing to do with taking a bath, but is derived from, and pronounced like, "to show".

Tuning penile tissue tone is the only realistic option for increasing penis size. For a flaccid state, proper penile tissue tone management will make more of a difference than for the erect state. For the flaccid state, some 70 percent of the size of the erect condition are within reach with natural phosphodiesterase type 5 inhibitors like Kaempferia parviflora and Boesenbergia rotunda (another member of the Zingiberaceae family).

Rigidity can be improved with yohimbe, but be warned: yohimbe can kill men with hypertension or a weak heart. yohimbe has been prohibited "in several European countries (e.g. United Kingdom, Ireland, Netherlands, Belgium, Denmark, Czech Republic) as well as in Canada, Australia and New Zealand." [Source: Evaluation of the safety of Yohimbe]. Even when yohimbe does not kill, side effect like tachycardia (a racing heart), heart palpitations (the sensation of a fluttering heart), and sleeplessness for up to 24 hours often make yohimbe an uncomfortable experience.

Don't want a bigger penis?

Even if you are not interested in a bigger penis, there are still good reasons to include Kaempferia parviflora in your diet.

You may want to run a search on scholar.google.com for: Kaempferia parviflora obesity

You will come across more than 20 studies, many of them double-blind and placebo-controlled, all pointing to the same result: methoxyflavonoids from Kaempferia parviflora inhibit the enzyme pancreatic lipase. Pancreatic lipase is released from the pancreas via ducts into the duodenum (the upper small intestine) where it hydrolyzes (= breaks of chemical bonds by water molecules) triglycerides, common in dietary fats. If pancreatic lipase is inhibited by Kaempferia parviflora, fewer lipids (fats) are taken up into the bloodstream, and thus there are fewer deposits of adipose tissue.

References:

2013, Scientific Opinion on the evaluation of the safety in use of Yohimbe (Pausinystalia yohimbe (K. Schum.) Pierre ex Beille), EFSA Journal, Vol 11(7), Pages 3302; https://efsa.onlinelibrary.wiley.com/doi/pdf/10.2903/j.efsa.2013.3302

Akase, T.; Shimada, T.; Terabayashi, S.; Ikeya, Y.; Sanada, H.; Aburada, M. (2011), Antiobesity effects of Kaempferia parviflora in spontaneously obese type 2 diabetic mice, Journal of Natural Medicines, Volume 65, Pages 73-80, 10.1007/s11418-010-0461-2

Asamenew, G.; Kim, H.-W.; Lee, M.-K.; Lee, S.-H.; Kim, Y.-J.; Cha,Y.-S.; Lim, J.-B. (2019) Characterization of phenolic compounds from normal ginger (Zingiber officinale Rosc.) and black ginger (Kaempferia parviflora Wall.) using UPLC–DAD–QToF–MS, European Food Research and Technology, Vol 245, Pages 653-665

Cartledge, J; Minhas, S; Eardley, I (2001), The role of nitric oxide in penile erection, Expert Opinion Pharmacotheraphy, Vol 2(1), Pages 95-107, https://doi.org/10.1517/14656566.2.1.95

Chaturapanich, G.; Chaiyakul, S.; Verawatnapakul, V.; Yimlamai, T.; Pholpramool, C.(2012), Enhancement of aphrodisiac activity in male rats by ethanol extract of Kaempferia parviflora and exercise training, Andrologia, Vol 44 (Suppl 1), Pages 323-328

Chaipech, S.; Morikawa, T.; Ninomiya, K.; Yoshikawa, M.l; Pongpiriyadacha, Y.; Hayakawa, T.; Muraoka, O. (2012), Structures of two new phenolic glycosides, kaempferiaosides A and B, and hepatoprotective constituents from the rhizomes of Kaempferia parviflora, Chemical and Pharmaceutical Bulletin, Vol 60, Pages 62-69

Deema P. (2007), Effect of Kaempferia parviflora and Endurance Training on Lactate Threshold in Humans Phitsanulok, Thailand: Naresuan University

Diastuti, Hartiwi; Chasani, Mochammad; Suwandri, Suwandri (2020), Antibacterial Activity of Benzyl Benzoate and Crotepoxide from Kaempferia rotunda L. Rhizome, Indonesian Journal of Chemistry, Vol 20, No 1

Grossman, E; Rosenthal, T; Peleg, E; Holmes, C; Goldstein, D S (1993), Oral yohimbine increases blood pressure and sympathetic nervous outflow in hypertensive patients, Journal Cadiovascular Pharmacology, Vol 22(1), Pages 22-6, https://doi.org/10.1097/00005344-199307000-00004

Hwang, Jae-Kwan; SA, Bo Kyung; Kim, Tae Yoon (2012), Use of kaempferia parviflora wall. ex. baker extracts or flavone compound for preventing or treating muscle diseases, or improving muscle function, Google Patents, https://patents.google.com/patent/EP2851071B1/en

Ghalayini, I F (2004), Nitric oxide–cyclic GMP pathway with some emphasis on cavernosal contractility, International Journal of Impotence Research, Volume 16, Pages 459–469, https://www.nature.com/articles/3901256

Klinjan, Preeyaporn; Mungkornasawakul, Pitchaya; Tragoolpua, Yingmanee; Sangthong, Padchanee (2021) Cytotoxicity and anti-herpes simplex virus type-1 activity of Kaempferia parviflora extract, APST, Vol. 26 No. 03

Kobayashi, Hiroko; Suzuki, Ryo; Sato, Kei; Ogami, Takatoshi; Tomozawa, Hiroshi; Tsubata, Masahito; Ichinose, Koji; Aburada, Masaki; Ochiai, Wataru; Sugiyama, Kiyoshi; Shimada, Tsutomu (2017), Effect of Kaempferia parviflora extract on knee osteoarthritis, Journal of Natural Medicines Volume 72, Pages 136–144

Kobayashi, Shoko; Kato, Taro; Azuma, Toshiaki; Kikuzaki, Hiroe; Abe, Keiko (2015), Anti-allergenic activity of polymethoxyflavones from Kaempferia parviflora, Journal of Functional Foods, Volume 13, Pages 100-107

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